Dysregulation of RNA methylation contributes to neurodegeneration
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Neuroscience
Link
https://www.nature.com/articles/s41593-023-01389-2.pdf
Reference5 articles.
1. Nussbacher, J. K., Tabet, R., Yeo, G. W. & Lagier-Tourenne, C. Disruption of RNA metabolism in neurological diseases andemerging therapeutic interventions. Neuron 102, 294–320 (2019). A review of neurological diseases with RNA metabolism dysregulation and the potential mechanisms.
2. Kim, G., Gautier, O., Tassoni-Tsuchida, E., Ma, X. R. & Gitler, A. D. ALS genetics: gains, losses, and implications for future therapies. Neuron 108, 822–842 (2020). A review of ALS genetics and disease mechanisms.
3. Balendra, R. & Isaacs, A. M. C9orf72-mediated ALS and FTD: multiple pathways to disease. Nat. Rev. Neurol. 14, 544–558 (2018). A review of pathogenetic mechanisms of C9orf72-mediated ALS and FTD.
4. Roundtree, I. A., Evans, M. E., Pan, T. & He, C. Dynamic RNA modifications in gene expression regulation. Cell 169, 1187–1200 (2017). A review of RNA modifications on coding and noncoding RNAs.
5. Cheng, W. et al. CRISPR-Cas9 screens identify the RNA helicase DDX3X as a repressor of C9ORF72 (GGGGCC)n repeat-associated non-AUG translation. Neuron 104, 885–898.e8 (2019). This paper reports CRISPR–Cas9 screens to identify modifiers of DPR protein production from C9orf72 (GGGGCC)n repeat expansion.
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