Premature polyadenylation-mediated loss of stathmin-2 is a hallmark of TDP-43-dependent neurodegeneration-Reference-Cited by-同舟云学术

Premature polyadenylation-mediated loss of stathmin-2 is a hallmark of TDP-43-dependent neurodegeneration

Published:2019-01-14 Issue:2 Volume:22 Page:180-190
ISSN:1097-6256
Container-title:Nature Neuroscience
language:en
Short-container-title:Nat Neurosci

Author:

Melamed Ze’ev,López-Erauskin Jone,Baughn Michael W.,Zhang Ouyang,Drenner Kevin,Sun Ying,Freyermuth Fernande,McMahon Moira A.,Beccari Melinda S.,Artates Jon W.,Ohkubo Takuya,Rodriguez Maria,Lin Nianwei,Wu Dongmei,Bennett C. Frank,Rigo Frank,Da Cruz Sandrine,Ravits John,Lagier-Tourenne Clotilde^ORCID,Cleveland Don W.^ORCID

Publisher

Springer Science and Business Media LLC

Subject

General Neuroscience

Link

http://www.nature.com/articles/s41593-018-0293-z.pdf

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3. DeJesus-Hernandez, M. et al. Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS. Neuron 72, 245–256 (2011).

4. Neumann, M. et al. Ubiquitinated TDP-43 in frontotemporal lobar degeneration and amyotrophic lateral sclerosis. Science 314, 130–133 (2006).

5. Ling, S. C., Polymenidou, M. & Cleveland, D. W. Converging mechanisms in ALS and FTD: disrupted RNA and protein homeostasis. Neuron 79, 416–438 (2013).

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