A non-Smad mechanism of fibroblast activation by transforming growth factor-β via c-Abl and Egr-1: selective modulation by imatinib mesylate-Reference-Cited by-同舟云学术

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A non-Smad mechanism of fibroblast activation by transforming growth factor-β via c-Abl and Egr-1: selective modulation by imatinib mesylate

Published:2009-01-19 Issue:10 Volume:28 Page:1285-1297
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Bhattacharyya S,Ishida W,Wu M,Wilkes M,Mori Y,Hinchcliff M,Leof E,Varga J

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2008479.pdf

Reference63 articles.

1. Bhattacharyya S, Chen S-J, Wu M, Warner-Blankenship M, Ning H, Lakos G et al. (2008). Smad-independent TGF-β regulation of transcription factor Egr-1 and sustained expression in fibrosis: implications for scleroderma. Am J Path 173: 1085–1099.

2. Buchwalter G, Gross C, Wasylyk B . (2004). Ets ternary complex transcription factors. Gene 324: 1–14.

3. Bonner JC . (2004). Regulation of PDGF and its receptors in fibrotic diseases. Cytokine Growth factor Rev 4: 255–273.

4. Ceni E, Crabb DW, Foschi M, Mello T, Taricchi M, Patussi V et al. (2006). Acetaldehyde inhibits PPAR-g via H2O2-mediated c-Abl activation in human hepatic stellate cells. Gastroenterology 131: 1235–1252.

5. Che W, Abe J, Yoshizumi M, Huang Q, Glassman M, Ohta S et al. (2001). p160 Bcr mediates platelet-derived growth factor activation of extracellular signal-regulated kinase in vascular smooth muscle cells. Circulation 104: 1399–1406.

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