Cellular and Molecular Mechanisms of Fibrosis in Systemic Sclerosis
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Publisher
Springer International Publishing
Link
https://link.springer.com/content/pdf/10.1007/978-3-031-40658-4_18
Reference233 articles.
1. Rockey DC, Bell PD, Hill JA. Fibrosis—a common pathway to organ injury and failure. N Engl J Med. 2015;372:1138–49. https://doi.org/10.1056/NEJMra1300575.
2. Ho YY, Lagares D, Tager AM, Kapoor M. Fibrosis—a lethal component of systemic sclerosis. Nat Rev Rheumatol. 2014;10:390–402. https://doi.org/10.1038/nrrheum.2014.53.
3. Rosenbloom J, Mendoza FA, Jimenez SA. Strategies for anti-fibrotic therapies. Biochim Biophys Acta. 2013;1832:1088–103. https://doi.org/10.1016/j.bbadis.2012.12.007.
4. Ramirez F, Tanaka S, Bou-Gharios G. Transcriptional regulation of the human alpha2(I) collagen gene (COL1A2), an informative model system to study fibrotic diseases. Matrix Biol. 2006;25:365–72. https://doi.org/10.1016/j.matbio.2006.05.002.
5. Ghosh AK, Wei J, Wu M, Varga J. Constitutive Smad signaling and Smad-dependent collagen gene expression in mouse embryonic fibroblasts lacking peroxisome proliferator-activated receptor-gamma. Biochem Biophys Res Commun. 2008;374:231–6. https://doi.org/10.1016/j.bbrc.2008.07.014.
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