Click editing enables programmable genome writing using DNA polymerases and HUH endonucleases-Reference-Cited by-同舟云学术

Click editing enables programmable genome writing using DNA polymerases and HUH endonucleases

Published:2024-07-22 Issue: Volume: Page:
ISSN:1087-0156
Container-title:Nature Biotechnology
language:en
Short-container-title:Nat Biotechnol

Author:

Ferreira da Silva Joana,Tou Connor J.^ORCID,King Emily M.^ORCID,Eller Madeline L.,Rufino-Ramos David^ORCID,Ma Linyuan,Cromwell Christopher R.,Metovic Jasna^ORCID,Benning Friederike M. C.^ORCID,Chao Luke H.^ORCID,Eichler Florian S.,Kleinstiver Benjamin P.^ORCID

Funder

U.S. Department of Health Human Services | NIH | NIH Office of the Director

Massachusetts General Hospital

U.S. Department of Health Human Services | NIH | National Heart, Lung, and Blood Institute

U.S. Department of Health Human Services | NIH | National Human Genome Research Institute

European Molecular Biology Organization

National Science Foundation

Friedreich's Ataxia Research Alliance

Banting Postdoctoral Fellowship - Natural Sciences and Engineering Research Council of Canada

Swiss National Science Foundation | National Center of Competence in Research Quantum Science and Technology

U.S. Department of Health Human Services | NIH | National Institute of General Medical Sciences

Publisher

Springer Science and Business Media LLC

Link

https://www.nature.com/articles/s41587-024-02324-x.pdf

Reference112 articles.

1. Doudna, J. A. The promise and challenge of therapeutic genome editing. Nature 578, 229–236 (2020).

2. Anzalone, A. V., Koblan, L. W. & Liu, D. R. Genome editing with CRISPR–Cas nucleases, base editors, transposases and prime editors. Nat. Biotechnol. 38, 824–844 (2020).

3. van Overbeek, M. et al. DNA repair profiling reveals nonrandom outcomes at Cas9-mediated breaks. Mol. Cell 63, 633–646 (2016).

4. Shen, M. W. et al. Predictable and precise template-free CRISPR editing of pathogenic variants. Nature 563, 646–651 (2018).

5. Kosicki, M., Tomberg, K. & Bradley, A. Repair of double-strand breaks induced by CRISPR–Cas9 leads to large deletions and complex rearrangements. Nat. Biotechnol. 36, 765–771 (2018).

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