N-acetyl Cysteine Treatment Rescues Cognitive Deficits Induced by Mitochondrial Dysfunction in G72/G30 Transgenic Mice-Reference-Cited by-同舟云学术

N-acetyl Cysteine Treatment Rescues Cognitive Deficits Induced by Mitochondrial Dysfunction in G72/G30 Transgenic Mice

Published:2011-06-29 Issue:11 Volume:36 Page:2233-2243
ISSN:0893-133X
Container-title:Neuropsychopharmacology
language:en
Short-container-title:Neuropsychopharmacol

Author:

Otte David-Marian,Sommersberg Britta,Kudin Alexei,Guerrero Catalina,Albayram Önder,Filiou Michaela D,Frisch Pamela,Yilmaz Öznur,Drews Eva,Turck Christoph W,Bilkei-Gorzó Andras,Kunz Wolfram S,Beck Heinz,Zimmer Andreas

Publisher

Springer Science and Business Media LLC

Subject

Psychiatry and Mental health,Pharmacology

Link

http://www.nature.com/articles/npp2011109.pdf

Reference87 articles.

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2. Addington AM, Gornick M, Sporn AL, Gogtay N, Greenstein D, Lenane M et al (2004). Polymorphisms in the 13q33.2 gene G72/G30 are associated with childhood-onset schizophrenia and psychosis not otherwise specified. Biol psychiatry 55: 976–980.

3. Altar CA, Jurata LW, Charles V, Lemire A, Liu P, Bukhman Y et al (2005). Deficient hippocampal neuron expression of proteasome, ubiquitin, and mitochondrial genes in multiple schizophrenia cohorts. Biol psychiatry 58: 85–96.

4. Andreazza AC, Shao L, Wang JF, Young LT (2010). Mitochondrial complex I activity and oxidative damage to mitochondrial proteins in the prefrontal cortex of patients with bipolar disorder. Arch Gen Psychiatry 67: 360–368.

5. Baker DA, McFarland K, Lake RW, Shen H, Toda S, Kalivas PW (2003). N-acetyl cysteine-induced blockade of cocaine-induced reinstatement. Ann N Y Acad Sci 1003: 349–351.

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