COX isoforms in the cardiovascular system: understanding the activities of non-steroidal anti-inflammatory drugs
Author:
Publisher
Springer Science and Business Media LLC
Subject
Drug Discovery,Pharmacology,General Medicine
Link
http://www.nature.com/articles/nrd1929.pdf
Reference128 articles.
1. Vane, J. R. Inhibition of prostaglandin synthesis as a mechanism of action for aspirin-like drugs. Nature New Biol. 231, 232–235 (1971).
2. Ferreira, S. H., Moncada, S. & Vane, J. R. Indomethacin and aspirin abolish prostaglandin release from the spleen. Nature New Biol. 231, 237–239 (1971).
3. Smith, J. B. & Willis, A. L. Aspirin selectively inhibits prostaglandin production in human platelets. Nature New Biol. 231, 235–237 (1971). References 1–3 are back-to-back papers establishing that NSAIDs have the common mechanism of action of inhibiting prostanoid formation. Reference 3 in particular showed that aspirin inhibited prostanoid formation in platelets; now known, of course, to be thromboxane A 2.
4. Vane, J. R., Bakhle, Y. S. & Botting, R. M. Cyclooxygenase 1 and 2. Annu. Rev. Pharmacol. Toxicol. 38, 97–120 (1998).
5. Smith, W. L., DeWitt, D. L. & Garavito, R. M. Cyclooxygenases: structural, cellular and molecular biology. Annu. Rev. Biochem. 69, 145–182 (2000).
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