Reversing EphB2 depletion rescues cognitive functions in Alzheimer model-Reference-Cited by-同舟云学术

Reversing EphB2 depletion rescues cognitive functions in Alzheimer model

Published:2010-11-28 Issue:7328 Volume:469 Page:47-52
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Cissé Moustapha,Halabisky Brian,Harris Julie,Devidze Nino,Dubal Dena B.,Sun Binggui,Orr Anna,Lotz Gregor,Kim Daniel H.,Hamto Patricia,Ho Kaitlyn,Yu Gui-Qiu,Mucke Lennart

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature09635.pdf

Reference50 articles.

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2. Shankar, G. M. et al. Amyloid-β protein dimers isolated directly from Alzheimer’s brains impair synaptic plasticity and memory. Nature Med. 14, 837–842 (2008)

3. Kamenetz, F. et al. APP processing and synaptic function. Neuron 37, 925–937 (2003)

4. Malenka, R. C. & Bear, M. F. LTP and LTD: an embarrassment of riches. Neuron 44, 5–21 (2004)

5. Ikonomovic, M. D. et al. Distribution of glutamate receptor subunit NMDAR1 in the hippocampus of normal elderly and patients with Alzheimer’s disease. Exp. Neurol. 160, 194–204 (1999)

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