Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory-Reference-Cited by-同舟云学术

Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

Published:2008-06-22 Issue:8 Volume:14 Page:837-842
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Shankar Ganesh M,Li Shaomin,Mehta Tapan H,Garcia-Munoz Amaya,Shepardson Nina E,Smith Imelda,Brett Francesca M,Farrell Michael A,Rowan Michael J,Lemere Cynthia A,Regan Ciaran M,Walsh Dominic M,Sabatini Bernardo L,Selkoe Dennis J

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm1782.pdf

Reference30 articles.

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3. Mucke, L. et al. High-level neuronal expression of Aβ 1–42 in wild-type human amyloid protein precursor transgenic mice: synaptotoxicity without plaque formation. J. Neurosci. 20, 4050–4058 (2000).

4. Morgan, D. et al. Aβ peptide vaccination prevents memory loss in an animal model of Alzheimer's disease. Nature 408, 982–985 (2000).

5. Walsh, D.M. et al. Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo. Nature 416, 535–539 (2002).

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