IL-17A and IL-17F in tissue homeostasis, inflammation and regeneration
Author:
Publisher
Springer Science and Business Media LLC
Subject
Rheumatology
Link
https://www.nature.com/articles/s41584-023-01004-5.pdf
Reference10 articles.
1. Adamopoulos, I. E. et al. IL-17A gene transfer induces bone loss and epidermal hyperplasia associated with psoriatic arthritis. Ann. Rheum. Dis. 74, 1284–1292 (2015).
2. Merola, J. F. et al. Bimekizumab in patients with active psoriatic arthritis and previous inadequate response or intolerance to tumour necrosis factor-α inhibitors: a randomised, double-blind, placebo-controlled, phase 3 trial (BE COMPLETE). Lancet 401, 38–48 (2023).
3. Gaublomme, J. T. et al. Single-cell genomics unveils critical regulators of TH17 cell pathogenicity. Cell 163, 1400–1412 (2015).
4. Su, Y. et al. Interleukin-17 receptor D constitutes an alternative receptor for interleukin-17A important in psoriasis-like skin inflammation. Sci. Immunol. 4, eaau9657 (2019).
5. Ono, T. et al. IL-17-producing γδ T cells enhance bone regeneration. Nat. Commun. 7, 10928 (2016).
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