Ketogenesis activates metabolically protective γδ T cells in visceral adipose tissue-Reference-Cited by-同舟云学术

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Ketogenesis activates metabolically protective γδ T cells in visceral adipose tissue

Published:2020-01 Issue:1 Volume:2 Page:50-61
ISSN:2522-5812
Container-title:Nature Metabolism
language:en
Short-container-title:Nat Metab

Author:

Goldberg Emily L.,Shchukina Irina,Asher Jennifer L.,Sidorov Sviatoslav,Artyomov Maxim N.^ORCID,Dixit Vishwa Deep

Funder

U.S. Department of Health & Human Services | NIH | National Institute on Aging

U.S. Department of Health & Human Services | NIH | National Institute of Arthritis and Musculoskeletal and Skin Diseases

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Physiology (medical),Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

http://www.nature.com/articles/s42255-019-0160-6.pdf

Reference64 articles.

1. Moreno, B. et al. Comparison of a very low-calorie-ketogenic diet with a standard low-calorie diet in the treatment of obesity. Endocrine 47, 793–805 (2014).

2. Moreno, B., Crujeiras, A. B., Bellido, D., Sajoux, I. & Casanueva, F. F. Obesity treatment by very low-calorie-ketogenic diet at two years: reduction in visceral fat and on the burden of disease. Endocrine 54, 681–690 (2016).

3. Shimazu, T. et al. Suppression of oxidative stress by beta-hydroxybutyrate, an endogenous histone deacetylase inhibitor. Science 339, 211–214 (2013).

4. Goldberg, E. L. et al. Beta-hydroxybutyrate deactivates neutrophil NLRP3 inflammasome to relieve gout flares. Cell Rep. 18, 2077–2087 (2017).

5. Lu, Y. et al. Ketogenic diet attenuates oxidative stress and inflammation after spinal cord injury by activating Nrf2 and suppressing the NF-kappaB signaling pathways. Neurosci. Lett. 683, 13–18 (2018).

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