Regulation of autoantibody activity by the IL-23–TH17 axis determines the onset of autoimmune disease-Reference-Cited by-同舟云学术

Regulation of autoantibody activity by the IL-23–TH17 axis determines the onset of autoimmune disease

Published:2016-11-07 Issue:1 Volume:18 Page:104-113
ISSN:1529-2908
Container-title:Nature Immunology
language:en
Short-container-title:Nat Immunol

Author:

Pfeifle René,Rothe Tobias,Ipseiz Natacha,Scherer Hans U,Culemann Stephan,Harre Ulrike,Ackermann Jochen A,Seefried Martina,Kleyer Arnd,Uderhardt Stefan,Haugg Benjamin,Hueber Axel J,Daum Patrick^ORCID,Heidkamp Gordon F,Ge Changrong,Böhm Sybille,Lux Anja,Schuh Wolfgang,Magorivska Iryna,Nandakumar Kutty S^ORCID,Lönnblom Erik,Becker Christoph,Dudziak Diana,Wuhrer Manfred,Rombouts Yoann^ORCID,Koeleman Carolien A,Toes René,Winkler Thomas H,Holmdahl Rikard^ORCID,Herrmann Martin^ORCID,Blüml Stephan^ORCID,Nimmerjahn Falk,Schett Georg,Krönke Gerhard

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/ni.3579.pdf

Reference41 articles.

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3. Grant, E.P. et al. Essential role for the C5a receptor in regulating the effector phase of synovial infiltration and joint destruction in experimental arthritis. J. Exp. Med. 196, 1461–1471 (2002).

4. Kleinau, S., Martinsson, P. & Heyman, B. Induction and suppression of collagen-induced arthritis is dependent on distinct Fcγ receptors. J. Exp. Med. 191, 1611–1616 (2000).

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