Histone deacetylase inhibitor but not arsenic trioxide differentiates acute promyelocytic leukaemia cells with t(11;17) in combination with all-transretinoic acid-Reference-Cited by-同舟云学术

Histone deacetylase inhibitor but not arsenic trioxide differentiates acute promyelocytic leukaemia cells with t(11;17) in combination with all-transretinoic acid

Published:2000-03-29 Issue:4 Volume:108 Page:696-702
ISSN:0007-1048
Container-title:British Journal of Haematology
language:en
Short-container-title:British Journal of Haematology

Author:

Kitamura Kunio,Hoshi Shinetsu,Koike Mitsuru,Kiyoi Hitoshi,Saito Hidehiko,Naoe Tomoki

Publisher

Wiley

Subject

Hematology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-2141.2000.01933.x/fullpdf

Reference41 articles.

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3. Use of arsenic trioxide (As2O3 ) in the treatment of acute promyelocytic leukemia (APL). I. As2O3 exerts dose-dependent dual effects on APL cells;Chen;Blood,1997

4. Rearrangements of the retinoic acid receptor alpha and promyelocytic leukemia zinc finger genes resulting from t (11; 17) (q23; q21) in a patient with acute promyelocytic leukemia;Chen;Journal of Clinical Investigation,1993

5. Fusion between a novel Krüppel-like zinc finger gene and the retinoic acid receptor-α locus due to a variant t (11; 17) translocation associated with acute promyelocytic leukemia;Chen;EMBO Journal,1993

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