Inhibition of T‐cell responsiveness by nasal peptide administration: influence of the thymus and differential recovery of T‐cell‐dependent functions
Author:
Affiliation:
1. Department of Pathology and Microbiology, University of Bristol School of Medical Sciences, University Walk, Bristol, UK
Publisher
Wiley
Subject
Immunology,Immunology and Allergy
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1046/j.1365-2567.1999.00795.x
Reference41 articles.
1. Inhibition of experimental autoimmune encephalomyelitis by inhalation but not oral administration of the encephalitogenic peptide: influence of MHC binding affinity
2. Mucosal Tolerance in a Murine Model of Experimental Autoimmune Encephalomyelitisa
3. Nasal administration of glutamate decarboxylase (GAD65) peptides induces Th2 responses and prevents murine insulin-dependent diabetes.
4. Protection of nonobese diabetic mice from diabetes by intranasal or subcutaneous administration of insulin peptide B-(9-23).
5. Mucosal tolerance and suppression of collagen-induced arthritis (CIA) induced by nasal inhalation of synthetic peptide 184-198 of bovine type II collagen (CII) expressing a dominant T cell epitope
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