Molecular, Genetic, and Cellular Pathogenesis of Neurofibromas and Surgical Implications
Author:
Affiliation:
1. Department of Neurosurgery, University of Utah, Salt Lake City, Utah
2. Department of Pediatrics, Division of Genetics, University of Utah, Salt Lake City, Utah
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Neurology (clinical),Surgery
Link
http://academic.oup.com/neurosurgery/article-pdf/58/1/1/24520352/neurosurgery-58-1-1.pdf
Reference210 articles.
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3. Mutations affecting mRNA splicing are the most common molecular defects in patients with neurofibromatosis type 1;Ars;Hum Mol Genet,2000
4. The NF1 tumor suppressor regulates mouse skin wound healing, fibroblast proliferation, and collagen deposited by fibroblasts;Atit;J Invest Dermatol,1999
5. Expression of Kit in neurofibromin-deficient human Schwann cells: Role in Schwann cell hyperplasia associated with type 1 neurofibromatosis;Badache;Oncogene,1998
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