Mechanisms of Acetaminophen-Induced Hepatotoxicity: Role of Oxidative Stress and Mitochondrial Permeability Transition in Freshly Isolated Mouse Hepatocytes-Reference-Cited by-同舟云学术

Mechanisms of Acetaminophen-Induced Hepatotoxicity: Role of Oxidative Stress and Mitochondrial Permeability Transition in Freshly Isolated Mouse Hepatocytes

Published:2004-10-01 Issue:2 Volume:312 Page:509-516
ISSN:0022-3565
Container-title:Journal of Pharmacology and Experimental Therapeutics
language:en
Short-container-title:J Pharmacol Exp Ther

Author:

Reid Angela B.,Kurten Richard C.,McCullough Sandra S.,Brock Robert W.,Hinson Jack A.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference38 articles.

1. Paracetamol (Acetaminophen)-Induced Toxicity: Molecular and Biochemical Mechanisms, Analogues and Protective Approaches

2. Role of Proinflammatory Cytokines in Acetaminophen Hepatotoxicity

3. Evidence for a direct role of intracellular calcium in paracetamol toxicity

4. Freshly isolated hepatocytes as a model for studying the toxicity of paracetamol

5. Selective Protein Covalent Binding and Target Organ Toxicity

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