Pharmacokinetic Analysis of the Blood-Brain Barrier Transport of 125I-Amyloid β Protein 40 in Wild-Type and Alzheimer's Disease Transgenic Mice (APP,PS1) and Its Implications for Amyloid Plaque Formation-Reference-Cited by-同舟云学术

Pharmacokinetic Analysis of the Blood-Brain Barrier Transport of 125I-Amyloid β Protein 40 in Wild-Type and Alzheimer's Disease Transgenic Mice (APP,PS1) and Its Implications for Amyloid Plaque Formation

Published:2005-03-02 Issue:3 Volume:313 Page:1370-1378
ISSN:0022-3565
Container-title:Journal of Pharmacology and Experimental Therapeutics
language:en
Short-container-title:J Pharmacol Exp Ther

Author:

Kandimalla Karunya K.,Curran Geoffry L.,Holasek Silvina S.,Gilles Emily J.,Wengenack Thomas M.,Poduslo Joseph F.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference27 articles.

1. In VivoAssessment of Brain Interstitial Fluid with Microdialysis Reveals Plaque-Associated Changes in Amyloid-β Metabolism and Half-Life

2. A Mathematical Model of the Impact of Novel Treatments on the AβBurden in the Alzheimer's Brain, CSF and Plasma

3. RAGE mediates amyloid-β peptide transport across the blood-brain barrier and accumulation in brain

4. Plaque-associated disruption of CSF and plasma amyloid-β (Aβ) equilibrium in a mouse model of Alzheimer's disease

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