Gβγ Inhibits Exocytosis via Interaction with Critical Residues on SolubleN-Ethylmaleimide-Sensitive Factor Attachment Protein-25-Reference-Cited by-同舟云学术

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Gβγ Inhibits Exocytosis via Interaction with Critical Residues on SolubleN-Ethylmaleimide-Sensitive Factor Attachment Protein-25

Published:2012-09-07 Issue:6 Volume:82 Page:1136-1149
ISSN:0026-895X
Container-title:Molecular Pharmacology
language:en
Short-container-title:Mol Pharmacol

Author:

Wells Christopher A.,Zurawski Zack,Betke Katherine M.,Yim Yun Young,Hyde Karren,Rodriguez Shelagh,Alford Simon,Hamm Heidi E.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference56 articles.

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2. Proteolysis of SNAP-25 by types E and A botulinal neurotoxins;Binz;J Biol Chem,1994

3. G protein βγ directly regulates SNARE protein fusion machinery for secretory granule exocytosis;Blackmer;Nat Neurosci,2005

4. G Protein βγ Subunit-Mediated Presynaptic Inhibition: Regulation of Exocytotic Fusion Downstream of Ca 2+ Entry

5. Synaptotagmin: a calcium sensor on the synaptic vesicle surface

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