Endothelium-Dependent Vasodilation in Human Mesenteric Artery Is Primarily Mediated by Myoendothelial Gap Junctions Intermediate Conductance Calcium-Activated K+ Channel and Nitric Oxide-Reference-Cited by-同舟云学术

Endothelium-Dependent Vasodilation in Human Mesenteric Artery Is Primarily Mediated by Myoendothelial Gap Junctions Intermediate Conductance Calcium-Activated K+ Channel and Nitric Oxide

Published:2010-12-20 Issue:3 Volume:336 Page:701-708
ISSN:0022-3565
Container-title:Journal of Pharmacology and Experimental Therapeutics
language:en
Short-container-title:J Pharmacol Exp Ther

Author:

Chadha Preet S.,Liu Lu,Rikard-Bell Matt,Senadheera Sevvandi,Howitt Lauren,Bertrand Rebecca L.,Grayson T. Hilton,Murphy Timothy V.,Sandow Shaun L.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference47 articles.

1. Phenotypic Heterogeneity of the Endothelium

2. Calcium‐activated potassium channel SK1‐ and IK1‐like immunoreactivity in injured human sensory neurones and its regulation by neurotrophic factors

3. Induces vasodilatation of rat mesenteric arteryin vitro mainly by inhibiting receptor-mediated Ca2+-influx and Ca2+-release

4. Obesity Up-Regulates Intermediate Conductance Calcium-Activated Potassium Channels and Myoendothelial Gap Junctions to Maintain Endothelial Vasodilator Function

5. Distinct hyperpolarizing and relaxant roles for gap junctions and endothelium-derived H2O2 in NO-independent relaxations of rabbit arteries

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