Pharmacologic Inhibition of the Renal Outer Medullary Potassium Channel Causes Diuresis and Natriuresis in the Absence of Kaliuresis-Reference-Cited by-同舟云学术

Pharmacologic Inhibition of the Renal Outer Medullary Potassium Channel Causes Diuresis and Natriuresis in the Absence of Kaliuresis

Published:2013-10-18 Issue:1 Volume:348 Page:153-164
ISSN:0022-3565
Container-title:Journal of Pharmacology and Experimental Therapeutics
language:en
Short-container-title:J Pharmacol Exp Ther

Author:

Garcia Maria L.,Priest Birgit T.,Alonso-Galicia Magdalena,Zhou Xiaoyan,Felix John P.,Brochu Richard M.,Bailey Timothy,Thomas-Fowlkes Brande,Liu Jessica,Swensen Andrew,Pai Lee-Yuh,Xiao Jianying,Hernandez Melba,Hoagland Kimberly,Owens Karen,Tang Haifeng,de Jesus Reynalda K.,Roy Sophie,Kaczorowski Gregory J.,Pasternak Alexander

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference26 articles.

1. Analgesic Effects of a Substituted N-Triazole Oxindole (TROX-1), a State-Dependent, Voltage-Gated Calcium Channel 2 Blocker

2. Development of a Selective Small-Molecule Inhibitor of Kir1.1, the Renal Outer Medullary Potassium Channel

3. Characterization of Kir1.1 Channels with the Use of a Radiolabeled Derivative of Tertiapin

4. The Inwardly Rectifying Potassium Channel Kir1.1: Development of Functional Assays to Identify and Characterize Channel Inhibitors

5. Functional Assay of Voltage-Gated Sodium Channels Using Membrane Potential-Sensitive Dyes

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