Small Molecules Selectively Targeting Sigma-1 Receptor for the Treatment of Neurological Diseases-Reference-Cited by-同舟云学术

Small Molecules Selectively Targeting Sigma-1 Receptor for the Treatment of Neurological Diseases

Published:2020-10-28 Issue:24 Volume:63 Page:15187-15217
ISSN:0022-2623
Container-title:Journal of Medicinal Chemistry
language:en
Short-container-title:J. Med. Chem.

Author:

Ye Na¹^ORCID,Qin Wangzhi¹,Tian Sheng¹,Xu Qingfeng¹,Wold Eric A.²^ORCID,Zhou Jia²^ORCID,Zhen Xue-Chu¹

Affiliation:

1. Jiangsu Key Laboratory of Neuropsychiatric Diseases and College of Pharmaceutical Sciences, Soochow University, Suzhou, Jiangsu 215123, China

2. Chemical Biology Program, Department of Pharmacology and Toxicology, and Center for Addiction Research, University of Texas Medical Branch, Galveston, Texas 77555, United States

Funder

Natural Science Foundation of Jiangsu Province

National Institute on Drug Abuse

University of Texas Medical Branch

National Natural Science Foundation of China

Priority Academic Program Development of Jiangsu Higher Education Institutions

Jiangsu Provincial Key Laboratory of Neuropsychiatric Diseases, Soochow University

Publisher

American Chemical Society (ACS)

Subject

Drug Discovery,Molecular Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.jmedchem.0c01192

Reference210 articles.

1. The sigma-1 receptor chaperone as an inter-organelle signaling modulator

2. Sigma-1 Receptor Activation Induces Autophagy and Increases Proteostasis Capacity In Vitro and In Vivo

3. Sigma-1 Receptor Agonists Induce Oxidative Stress in Mitochondria and Enhance Complex I Activity in Physiological Condition but Protect Against Pathological Oxidative Stress

4. Dysfunction in endoplasmic reticulum-mitochondria crosstalk underlies SIGMAR1 loss of function mediated motor neuron degeneration

5. Genetic Inactivation and Pharmacological Blockade of Sigma-1 Receptors Prevent Paclitaxel-Induced Sensory-Nerve Mitochondrial Abnormalities and Neuropathic Pain in Mice

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