Immunostimulation of Fibrous Nucleic Acid Nanoparticles Can be Modulated through Aptamer-Based Functional Moieties: Unveiling the Structure–Activity Relationship and Mechanistic Insights-Reference-Cited by-同舟云学术

Immunostimulation of Fibrous Nucleic Acid Nanoparticles Can be Modulated through Aptamer-Based Functional Moieties: Unveiling the Structure–Activity Relationship and Mechanistic Insights

Published:2024-02-12 Issue:7 Volume:16 Page:8430-8441
ISSN:1944-8244
Container-title:ACS Applied Materials & Interfaces
language:en
Short-container-title:ACS Appl. Mater. Interfaces

Author:

Rebolledo Laura P.¹,Ke Weina¹,Cedrone Edward²,Wang Jian³^ORCID,Majithia Krishna⁴,Johnson M. Brittany⁴,Dokholyan Nikolay V.³⁵^ORCID,Dobrovolskaia Marina A.²^ORCID,Afonin Kirill A.¹^ORCID

Affiliation:

1. Nanoscale Science Program, Department of Chemistry, University of North Carolina Charlotte, Charlotte, North Carolina 28223, United States

2. Nanotechnology Characterization Laboratory, Cancer Research Technology Program, Frederick National Laboratory for Cancer Research Sponsored by the National Cancer Institute, Frederick, Maryland 21701, United States

3. Department of Pharmacology, Penn State College of Medicine, Hershey, Pennsylvania 17033, United States

4. Department of Biological Sciences, University of North Carolina Charlotte, Charlotte, North Carolina 28223, United States

5. Department of Biochemistry & Molecular Biology, Department of Biochemistry & Molecular Biology, Penn State College of Medicine, Hershey, Pennsylvania 17033, United States

Funder

National Cancer Institute

National Institute of General Medical Sciences

Publisher

American Chemical Society (ACS)

Link

https://pubs.acs.org/doi/pdf/10.1021/acsami.3c17779

Reference80 articles.

1. Titanium dioxide nanofibers induce angiogenic markers and genomic instability in lung cells leading to a highly dedifferentiated and fibrotic tumor formation in a xenograft model

2. Nanoparticles as a Potential Cause of Pleural and Interstitial Lung Disease

3. The significance of nanoparticles in particle- induced pulmonary fibrosis

4. Comparison of toxicity and cellular responses following pulmonary exposure to different types of nanofibers

5. Carbon nanotubes activate inflammatory signalling through binding to Siglec-14

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