Calmodulin Mediates DNA Repair Pathways Involving H2AX in Response to Low-Dose Radiation Exposure of RAW 264.7 Macrophages-Reference-Cited by-同舟云学术

Calmodulin Mediates DNA Repair Pathways Involving H2AX in Response to Low-Dose Radiation Exposure of RAW 264.7 Macrophages

Published:2009-02-05 Issue:3 Volume:22 Page:460-470
ISSN:0893-228X
Container-title:Chemical Research in Toxicology
language:en
Short-container-title:Chem. Res. Toxicol.

Author:

Smallwood Heather S.¹,Lopez-Ferrer Daniel¹,Eberlein P. Elis¹,Watson David J.¹,Squier Thomas C.¹

Affiliation:

1. Washington State University Tri-Cities, Richland, Washington 99352, and Cell Biology and Biochemistry Group, Biological Sciences Division, Pacific Northwest National Laboratory, Richland, Washington 99352

Publisher

American Chemical Society (ACS)

Subject

Toxicology,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/tx800236r

Reference71 articles.

1. Diminished Expression of Transcription Factors Nuclear Factor κB and CCAAT/Enhancer Binding Protein Underlies a Novel Tumor Evasion Mechanism Affecting Macrophages of Mammary Tumor–Bearing Mice

2. Role of tumor-associated macrophages in tumor progression and invasion

3. The NF-κB/IκB signaling system: A molecular target in breast cancer therapy

4. Low-Dose Hyper-radiosensitivity: A Consequence of Ineffective Cell Cycle Arrest of Radiation-Damaged G2-Phase Cells

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