The 2-Deoxyribonolactone Lesion Produced in DNA by Neocarzinostatin and Other Damaging Agents Forms Cross-links with the Base-Excision Repair Enzyme Endonuclease III-Reference-Cited by-同舟云学术

The 2-Deoxyribonolactone Lesion Produced in DNA by Neocarzinostatin and Other Damaging Agents Forms Cross-links with the Base-Excision Repair Enzyme Endonuclease III

Published:2001-03-09 Issue:13 Volume:123 Page:3161-3162
ISSN:0002-7863
Container-title:Journal of the American Chemical Society
language:en
Short-container-title:J. Am. Chem. Soc.

Author:

Hashimoto Mitsumasa¹,Greenberg Marc M.¹,Kow Yoke W.¹,Hwang Jae-Taeg¹,Cunningham Richard P.¹

Affiliation:

1. Division of Cancer Biology, Department of Radiation Oncology, Emory University School of Medicine Atlanta, Georgia 30335 Department of Chemistry, Colorado State University Fort Collins, Colorado 80523 Department of Biological Sciences, SUNY at Albany Albany, New York 12222

Publisher

American Chemical Society (ACS)

Subject

Colloid and Surface Chemistry,Biochemistry,General Chemistry,Catalysis

Link

https://pubs.acs.org/doi/pdf/10.1021/ja003354z

Reference20 articles.

1. Chemistry of Glycosylases and Endonucleases Involved in Base-Excision Repair

2. Mechanism-Based DNA−Protein Cross-Linking of MutY via Oxidation of 8-Oxoguanosine

3. Investigating Nucleic Acid Damage Processes via Independent Generation of Reactive Intermediates

4. Kinetics and Stereoselectivity of Thiol Trapping of Deoxyuridin-1‘-yl in Biopolymers and Their Relationship to the Formation of Premutagenic α-Deoxynucleotides

5. Highly sequence-selective photoreaction of 5-bromouracil-containing deoxyhexanucleotides

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