Intracellular Formation of a DNA Damage-Induced, Histone Post-Translational Modification Following Bleomycin Treatment
Author:
Affiliation:
1. Department of Chemistry, Johns Hopkins University, 3400 N. Charles Street, Baltimore, Maryland 21218, United States
2. Thomas C. Jenkins Department of Biophysics, Johns Hopkins University, Baltimore, Maryland 21218, United States
Funder
National Institute of General Medical Sciences
Publisher
American Chemical Society (ACS)
Subject
Colloid and Surface Chemistry,Biochemistry,General Chemistry,Catalysis
Link
https://pubs.acs.org/doi/pdf/10.1021/jacs.2c02880
Reference54 articles.
1. Implications of DNA damage and DNA repair on human diseases
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3. Formation and repair of unavoidable, endogenous interstrand cross-links in cellular DNA
4. Irreversible Inhibition of DNA Polymerase β by an Oxidized Abasic Lesion
5. The 2-Deoxyribonolactone Lesion Produced in DNA by Neocarzinostatin and Other Damaging Agents Forms Cross-links with the Base-Excision Repair Enzyme Endonuclease III
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