Staphylococcus aureus β-Toxin Mutants Are Defective in Biofilm Ligase and Sphingomyelinase Activity, and Causation of Infective Endocarditis and Sepsis-Reference-Cited by-同舟云学术

Staphylococcus aureus β-Toxin Mutants Are Defective in Biofilm Ligase and Sphingomyelinase Activity, and Causation of Infective Endocarditis and Sepsis

Published:2016-04-15 Issue:17 Volume:55 Page:2510-2517
ISSN:0006-2960
Container-title:Biochemistry
language:en
Short-container-title:Biochemistry

Author:

Herrera Alfa¹,Vu Bao G.¹,Stach Christopher S.¹,Merriman Joseph A.¹,Horswill Alexander R.¹,Salgado-Pabón Wilmara¹,Schlievert Patrick M.¹

Affiliation:

1. Department of Microbiology, University of Iowa Carver College of Medicine, Iowa City, Iowa 52242, United States

Publisher

American Chemical Society (ACS)

Subject

Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.biochem.6b00083

Reference24 articles.

1. Staphylococcus aureus β-toxin Production is Common in Strains With the β-toxin Gene Inactivated by Bacteriophage

2. Beta toxin catalyzes formation of nucleoprotein matrix in staphylococcal biofilms

3. Beta-Hemolysin Promotes Skin Colonization by Staphylococcus aureus

4. Comparison of Staphylococcus aureus strains for ability to cause infective endocarditis and lethal sepsis in rabbits

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