Molecular Insights into Human Hereditary Apolipoprotein A-I Amyloidosis Caused by the Glu34Lys Mutation-Reference-Cited by-同舟云学术

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Molecular Insights into Human Hereditary Apolipoprotein A-I Amyloidosis Caused by the Glu34Lys Mutation

Published:2018-09-05 Issue:39 Volume:57 Page:5738-5747
ISSN:0006-2960
Container-title:Biochemistry
language:en
Short-container-title:Biochemistry

Author:

Morgado Isabel¹,Panahi Afra²,Burwash Andrew G.²,Das Madhurima¹,Straub John E.²,Gursky Olga¹³^ORCID

Affiliation:

1. Department of Physiology & Biophysics, Boston University School of Medicine, Boston, Massachusetts 02118-2526, United States

2. Department of Chemistry, Boston University, Boston, Massachusetts 02215-2521, United States

3. Amyloidosis Treatment and Research Center, Boston University School of Medicine, Boston, Massachusetts 02118, United States

Funder

H2020 Marie Sklodowska-Curie Actions

National Institute of General Medical Sciences

Stewart Amyloidosis Research Fund

Publisher

American Chemical Society (ACS)

Subject

Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.biochem.8b00817

Reference37 articles.

1. Variant apolipoprotein AI as a major constituent of a human hereditary amyloid

2. Apolipoproteins and amyloid fibril formation in atherosclerosis

3. Amyloid-Forming Properties of Human Apolipoproteins: Sequence Analyses and Structural Insights

4. Cholesterol Efflux and Atheroprotection

5. Dysfunctional HDL containing L159R ApoA-I leads to exacerbation of atherosclerosis in hyperlipidemic mice

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1. The Apparent Organ-Specificity of Amyloidogenic ApoA-I Variants Is Linked to Tissue-Specific Extracellular Matrix Components;International Journal of Molecular Sciences;2022-12-24

2. Leveraging knowledge of HDLs major protein ApoA1: Structure, function, mutations, and potential therapeutics;Biomedicine & Pharmacotherapy;2022-10

3. Autophagy Alteration in ApoA-I Related Systemic Amyloidosis;International Journal of Molecular Sciences;2022-03-23

4. Molecular simulation unravels the amyloidogenic misfolding of nascent ApoA1 protein, driven by deleterious point mutations occurring in between 170–178 hotspot region;Journal of Biomolecular Structure and Dynamics;2021-10-06

5. Hereditary Apolipoprotein A-1 Amyloidosis With Glu34Lys Mutation Treated by Liver Transplantation: A Case Report;Transplantation Proceedings;2021-05

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