Dysfunctional HDL containing L159R ApoA-I leads to exacerbation of atherosclerosis in hyperlipidemic mice-Reference-Cited by-同舟云学术

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Dysfunctional HDL containing L159R ApoA-I leads to exacerbation of atherosclerosis in hyperlipidemic mice

Published:2012-03 Issue:3 Volume:1821 Page:502-512
ISSN:1388-1981
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids

Author:

Sorci-Thomas Mary G.,Zabalawi Manal,Bharadwaj Manish S.,Wilhelm Ashley J.,Owen John S.,Asztalos Bela F.,Bhat Shaila,Thomas Michael J.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference53 articles.

1. High density lipoprotein as a protective factor against coronary heart disease. The Framingham Study;Gordon;Am. J. Med.,1977

2. ApoA-I deficiency causes both hypertriglyceridemia and increased atherosclerosis in human apoB transgenic mice;Voyiaziakis;J. Lipid Res.,1998

3. Inhibition of early atherogenesis in transgenic mice by human apolipoprotein A-I;Rubin;Nature,1991

4. High-density lipoproteins and atherosclerosis;Rader;Am. J. Cardiol.,2002

5. The Tangier disease gene product ABC1 controls the cellular apolipoprotein-mediated lipid removal pathway;Lawn;J. Clin. Invest.,1999

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