Phospholipid Hydrolysis Caused by Clostridium perfringens α-Toxin Facilitates the Targeting of Perfringolysin O to Membrane Bilayers
Author:
Affiliation:
1. Department of Biochemistry and Molecular Biology, University of Massachusetts, Amherst, Massachusetts 01003, United States
Publisher
American Chemical Society (ACS)
Subject
Biochemistry
Link
https://pubs.acs.org/doi/pdf/10.1021/bi1013886
Reference90 articles.
1. Molecular and Cellular Basis of Microvascular Perfusion Deficits Induced by Clostridium perfringens and Clostridium septicum
2. Clostridial toxins in the pathogenesis of gas gangrene
3. Virulence studies on chromosomal ?-toxin and ?-toxin mutants constructed by allelic exchange provide genetic evidence for the essential role of ?-toxin in Clostridium perfringens-mediated gas gangrene
4. Alpha toxin from Clostridium perfringens induces proinflammatory changes in endothelial cells.
5. Clostridium perfringens α-toxin induces rabbit neutrophil adhesion
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