Glutathione-Deficient Mice Are Susceptible to TCDD-Induced Hepatocellular Toxicity but Resistant to Steatosis-Reference-Cited by-同舟云学术

Glutathione-Deficient Mice Are Susceptible to TCDD-Induced Hepatocellular Toxicity but Resistant to Steatosis

Published:2011-12-06 Issue:1 Volume:25 Page:94-100
ISSN:0893-228X
Container-title:Chemical Research in Toxicology
language:en
Short-container-title:Chem. Res. Toxicol.

Author:

Chen Ying¹,Krishan Mansi²,Nebert Daniel W.²,Shertzer Howard G.²

Affiliation:

1. Department of Pharmaceutical Sciences, School of Pharmacy, University of Colorado Denver, Aurora, Colorado 80045, United States

2. Department of Environmental Health and Center for Environmental Genetics, University of Cincinnati Medical Center, P.O. Box 670056, Cincinnati, Ohio 45267, United States

Publisher

American Chemical Society (ACS)

Subject

Toxicology,General Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/tx200242a

Reference47 articles.

1. Regulation of hepatic glutathione metabolism and its role in hepatotoxicity

2. Glutathione deficiency produced by inhibition of its synthesis, and its reversal; Applications in research and therapy

3. Genetically altered mice to evaluate glutathione homeostasis in health and disease

4. Glutamate Cysteine Ligase Catalysis

5. Hepatocyte-specificGclcdeletion leads to rapid onset of steatosis with mitochondrial injury and liver failure

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