Oxidative stress and genotoxicity in 1,4-dioxane liver toxicity as evidenced in a mouse model of glutathione deficiency-Reference-Cited by-同舟云学术

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Oxidative stress and genotoxicity in 1,4-dioxane liver toxicity as evidenced in a mouse model of glutathione deficiency

Published:2022-02 Issue: Volume:806 Page:150703
ISSN:0048-9697
Container-title:Science of The Total Environment
language:en
Short-container-title:Science of The Total Environment

Author:

Chen Ying,Wang Yewei,Charkoftaki Georgia,Orlicky David J.,Davidson Emily,Wan Fengjie,Ginsberg Gary,Thompson David C.,Vasiliou Vasilis

Funder

National Institutes of Health

Publisher

Elsevier BV

Subject

Pollution,Waste Management and Disposal,Environmental Chemistry,Environmental Engineering

Reference52 articles.

1. 1,4-Dioxane drinking water occurrence data from the third unregulated contaminant monitoring rule;Adamson;Sci. Total Environ.,2017

2. Toxicological Profile for 1,4 Dioxane;ATSDR,2012

3. Support Document to the 2017 Substance Priority List (Candidates for Toxicological Profiles);ATSDR,2017

4. Oxidative stress, toxicology, and pharmacology of CYP2E1;Caro;Annu.Rev.Pharmacol.Toxicol.,2004

5. CYP2E1 sensitizes the liver to LPS- and TNF alpha-inducedtoxicity via elevatedoxidative and nitrosativestress and activation of ASK-1 and JNK mitogen-activatedkinases;Cederbaum;Int. J. Hepatol.,2012

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