Isoniazid Oxidation by Mycobacterium tuberculosis KatG: A Role for Superoxide Which Correlates with Isoniazid Susceptibility-Reference-Cited by-同舟云学术

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Isoniazid Oxidation by Mycobacterium tuberculosis KatG: A Role for Superoxide Which Correlates with Isoniazid Susceptibility

Published:1999-10-01 Issue:41 Volume:121 Page:9748-9749
ISSN:0002-7863
Container-title:Journal of the American Chemical Society
language:en
Short-container-title:J. Am. Chem. Soc.

Author:

Wengenack Nancy L.¹,Hoard Heidi M.¹,Rusnak Frank¹

Affiliation:

1. Department of Biochemistry and Molecular Biology and Section of Hematology Research Mayo Clinic and Foundation, Rochester, Minnesota 55905

Publisher

American Chemical Society (ACS)

Subject

Colloid and Surface Chemistry,Biochemistry,General Chemistry,Catalysis

Link

https://pubs.acs.org/doi/pdf/10.1021/ja992590a

Reference30 articles.

1. World Health Organization, 1998.

2. Studies on the Mechanism of Action of Isoniazid and Ethionamide in the Chemotherapy of Tuberculosis

3. Mechanistic Studies of the Oxidation of Isoniazid by the Catalase Peroxidase from Mycobacterium tuberculosis

4. Missense mutations in the catalase-peroxidase gene, katG, are associated with isoniazid resistance in Mycobacterium tuberculosis

5. Characterization of the Catalase-Peroxidase Gene (katG) and inhA Locus in Isoniazid-Resistant and -Susceptible Strains of Mycobacterium tuberculosis by Automated DNA Sequencing: Restricted Array of Mutations Associated with Drug Resistance

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