Abstract
The current view on the cause of the non-coagulability of the blood after the injection of “peptone” is that the injected material stimulates the liver (or the liver plus other organs) to form an excess of a substance named antithrombin. The liver is considered further to form antithrombin under normal conditions in amount sufficient to maintain the fluidity of the blood. The statement that “peptone” which follows its slow injection is commonly ascribed to hepatic activity. The experiments recorded in this paper show that these views can no longer be held.
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2 articles.
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1. Blood coagulation, anti-coagulants and hæmolysis;Proceedings of the Royal Society of London. Series B, Containing Papers of a Biological Character;1924-10
2. The action of “peptone” and of nucleic acids on the coagulability of the blood;Proceedings of the Royal Society of London. Series B, Containing Papers of a Biological Character;1924-02