Evaluation of trigger factors of acute encephalopathy in glutaric aciduria type I: Fever and tumour necrosis factor-α

Author:

Kölker S.1,Ahlmeyer B.2,Krieglstein J.2,Hoffmann G. F.1

Affiliation:

1. ; Department of Neuropediatrics and Metabolic Diseases; University Children's Hospital; Marburg Germany

2. ; Department of Pharmacology and Toxicology; Philipps University of Marburg; Germany

Publisher

Wiley

Subject

Genetics (clinical),Genetics

Reference7 articles.

1. Tumor necrosis factor-alpha potentiates glutamate neurotoxicity in human fetal brain cell cultures;Chao;Dev Neurosci,1994

2. The transcription factor NF-iB mediates increases in calcium currents and decreases in NMDA-and AMPA/kainate-induced currents induced by tumor necrosis factor-a in hippocampal neurons;Furukawa;J Neurosci,1998

3. Ischemic stroke: From basic mechanism to new drug development;Ginsberg;Monogr Clin Neurosci,1998

4. Clinical course, early diagnosis, treatment and prevention of disease in glutaryl-CoA dehydrogenase deficiency;Ho.mann;Neuropediatrics,1996

5. 3-Hydroxyglutaric and glutaric acids are neurotoxic through NMDA receptors in vitro;Kölker;J Inherit Metab Dis,1999

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