Participation of neuronal nitric oxide synthase in experimental neuropathic pain induced by sciatic nerve transection

Author:

Chacur M.1,Matos R.J.B.2,Alves A.S.2,Rodrigues A.C.3,Gutierrez V.4,Cury Y.4,Britto L.R.G.2

Affiliation:

1. Universidade de São Paulo; Universidade de São Paulo

2. Universidade de São Paulo

3. Universidade de São Paulo, Brasil

4. Instituto Butantan, Brasil

Publisher

FapUNIFESP (SciELO)

Subject

Cell Biology,General Pharmacology, Toxicology and Pharmaceutics,General Medicine,Immunology,Physiology,Biochemistry,General Neuroscience,Biophysics

Reference34 articles.

1. Production of endogenous nitric oxide and activation of soluble guanylate cyclase are required for N-methyl-D-aspartate-produced facilitation of the nociceptive tail-flick reflex;Meller ST;Eur J Pharmacol,1992

2. Nitric oxide: action and pathological roles;Dawson TM;Neuroscientist,1995

3. Expression of neuronal isoform of nitric oxide synthase in spinal neurons of neonatal rats after sciatic nerve transection;Rogerio F;Neurosci Lett,2001

4. The effect of N-nitro-L-arginine and aminoguanidine treatment on changes in constitutive and inducible nitric oxide synthases in the spinal cord after sciatic nerve transection;Lukacova N;Int J Mol Med,2008

5. Nitric oxide mediates behavioral signs of neuropathic pain in an experimental rat model;Yoon YW;Neuroreport,1998

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