Mitochondrial Dysfunction in Alzheimer’s Disease and the Rationale for Bioenergetics Based Therapies
Author:
Publisher
Aging and Disease
Subject
Cell Biology,Clinical Neurology,Geriatrics and Gerontology,Pathology and Forensic Medicine
Link
http://www.aginganddisease.org/fileup/2152-5250/PDF/2152-5250-2016-2-201.pdf
Reference205 articles.
1. Swerdlow RH, Burns JM, Khan SM (2014). The Alzheimer's disease mitochondrial cascade hypothesis: progress and perspectives. Biochim Biophys Acta, 1842:1219-1231
2. Fukui H, Moraes CT (2008). The mitochondrial impairment, oxidative stress and neurodegeneration connection: reality or just an attractive hypothesis? Trends Neurosci, 31:251-256
3. Picone P, Nuzzo D, Caruana L, Scafidi V, Di Carlo M (2014). Mitochondrial Dysfunction: Different Routes to Alzheimer’s Disease Therapy. Oxidative Medicine and Cellular Longevity. Oxid Med Cell Longev, 2014:780179.
4. Scarpulla RC (2008). Transcriptional paradigms in mammalian mitochondrial biogenesis and function. Physiol Rev, 88: 611-638.
5. Mosconi L, Brys M, Switalski R, Mistur R, Glodzik L, Pirraglia E,et al. (2007). Maternal family history of Alzheimer's disease predisposes to reduced brain glucose metabolism. Proc Natl Acad Sci U S A, 104: 19067-19072.
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