Omi/HtrA2 Participates in Age-Related Autophagic Deficiency in Rat Liver-Reference-Cited by-同舟云学术

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Omi/HtrA2 Participates in Age-Related Autophagic Deficiency in Rat Liver

Published:2018 Issue:6 Volume:9 Page:1031
ISSN:2152-5250
Container-title:Aging and disease
language:en
Short-container-title:Aging and disease

Author:

Xu Jiahui,Jiao Kun,Liu Xin,Sun Qi,Wang Ke,Xu Haibo,Zhang Shangyue,Wu Ye,Wu Linguo,Liu Dan,Wang Wen,Liu Huirong

Publisher

Aging and Disease

Subject

Cell Biology,Neurology (clinical),Geriatrics and Gerontology,Pathology and Forensic Medicine

Link

http://www.aginganddisease.org/fileup/2152-5250/PDF/ad-0-0-0-1803050920-1.pdf

Reference42 articles.

1. Lipinska B, Fayet O, Baird L, Georgopoulos C (1989). Identification, characterization, and mapping of the Escherichia coli htrA gene, whose product is essential for bacterial growth only at elevated temperatures. J Bacteriol, 171:1574–84.

2. Faccio L, Fusco C, Chen A, Martinotti S, Bonventre JV, Zervos AS (2000). Characterization of a novel human serine protease that has extensive homology to bacterial heat shock endoprotease HtrA and is regulated by kidney ischemia. J Biol Chem, 275(4):2581–8.

3. Hegde R, Srinivasula SM, Zhang Z, Wassell R, Mukattash R, Cilenti L, et al. (2002). Identification of Omi/HtrA2 as a mitochondrial apoptotic serine protease that disrupts inhibitor of apoptosis protein-caspase interaction. J Biol Chem, 277: 432–438.

4. Dagda RK, Chu CT (2009). Mitochondrial quality control: insights on how Parkinson's disease related genes PINK1, parkin, and Omi/HtrA2 interact to maintain mitochondrial homeostasis. J Bioenerg Biomembr, 41:473–9.

5. Jones JM, Datta P, Srinivasula SM, Ji W, Gupta S, Zhang Z, et al. (2003). Loss of Omi mitochondrial protease activity causes the neuromuscular disorder of mnd2 mutant mice. Nature, 425: 721–727.

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