LMNA-mutated Rabbits: A Model of Premature Aging Syndrome with Muscular Dystrophy and Dilated Cardiomyopathy-Reference-Cited by-同舟云学术

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LMNA-mutated Rabbits: A Model of Premature Aging Syndrome with Muscular Dystrophy and Dilated Cardiomyopathy

Published:2019 Issue:1 Volume:10 Page:102
ISSN:2152-5250
Container-title:Aging and disease
language:en
Short-container-title:Aging and disease

Author:

Sui Tingting,Liu Di,Liu Tingjun,Deng Jichao,Chen Mao,Xu Yuanyuan,Song Yuning,Ouyang Hongsheng,Lai Liangxue,Li Zhanjun

Publisher

Aging and Disease

Subject

Cell Biology,Neurology (clinical),Geriatrics and Gerontology,Pathology and Forensic Medicine

Link

http://www.aginganddisease.org/fileup/2152-5250/PDF/ad-0-0-0-1802161100-1.pdf

Reference50 articles.

1. Lee DC, Welton KL, Smith ED, Kennedy BK (2009). A-type nuclear lamins act as transcriptional repressors when targeted to promoters. Exp Cell Res, 315:996-1007.

2. Shumaker DK, Solimando L, Sengupta K, Shimi T, Adam SA, Grunwald A, et al. (2008). The highly conserved nuclear lamin Ig-fold binds to PCNA: its role in DNA replication. J Cell Biol, 181:269-280.

3. Spann TP, Goldman AE, Wang C, Huang S, Goldman RD (2002). Alteration of nuclear lamin organization inhibits RNA polymerase II-dependent transcription. J Cell Biol, 156:603-608.

4. Spann TP, Moir RD, Goldman AE, Stick R, Goldman RD (1997). Disruption of nuclear lamin organization alters the distribution of replication factors and inhibits DNA synthesis. J Cell Biol, 136:1201-1212.

5. Moir RD, Spann TP, Goldman RD (1995). The dynamic properties and possible functions of nuclear lamins. Int Rev Cytol, 162B:141-182.

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