Diaphragm Abnormalities in Patients with End-Stage Heart Failure: NADPH Oxidase Upregulation and Protein Oxidation-Reference-Cited by-同舟云学术

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Diaphragm Abnormalities in Patients with End-Stage Heart Failure: NADPH Oxidase Upregulation and Protein Oxidation

Published:2017-01-09 Issue: Volume:7 Page:
ISSN:1664-042X
Container-title:Frontiers in Physiology
language:
Short-container-title:Front. Physiol.

Author:

Ahn Bumsoo,Coblentz Philip D.,Beharry Adam W.,Patel Nikhil,Judge Andrew R.,Moylan Jennifer. S.,Hoopes Charles W.,Bonnell Mark R.,Ferreira Leonardo F.

Funder

National Heart, Lung, and Blood Institute

National Institutes of Health

Publisher

Frontiers Media SA

Subject

Physiology (medical),Physiology

Reference54 articles.

1. NAD(P)H oxidase subunit p47phox is elevated, and p47phox knockout prevents diaphragm contractile dysfunction in heart failure;Ahn;Am. J. Physiol. Lung Cell Mol. Physiol.,2015

2. Breathing pattern, ventilatory drive and respiratory muscle strength in patients with chronic heart failure;Ambrosino;Eur. Respir. J.,1994

3. The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology;Bedard;Physiol. Rev.,2007

4. Oxygen free radicals and congestive heart failure;Belch;Br Heart J.,1991

5. Trolox attenuates mechanical ventilation-induced diaphragmatic dysfunction and proteolysis;Betters;Am. J. Respir. Crit. Care Med.,2004

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4. Skeletal muscle Nox4 knockout prevents and Nox2 knockout blunts loss of maximal diaphragm force in mice with heart failure with reduced ejection fraction;Free Radical Biology and Medicine;2023-01

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