TRAP1: A Metabolic Hub Linking Aging Pathophysiology to Mitochondrial S-Nitrosylation
Author:
Funder
Kræftens Bekæmpelse
Novo Nordisk Fonden
Publisher
Frontiers Media SA
Subject
Physiology (medical),Physiology
Reference38 articles.
1. Mitochondria in the signaling pathways that control longevity and health span;Akbari;Ageing Res. Rev.,2019
2. TRAP1 revisited: novel localizations and functions of a “next-generation” biomarker (Review);Amoroso;Int. J. Oncol.,2014
3. Mitochondrial chaperone TRAP1 activates the mitochondrial UPR and extends healthspan in Drosophila;Baqri;Mech. Ageing Dev.,2014
4. The mitochondrial chaperone protein TRAP1 mitigates α-synuclein toxicity;Butler;PLoS Genet.,2012
5. S-nitrosylation of parkin regulates ubiquitination and compromises parkin's protective function;Chung;Science,2004
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1. Parkinson's disease gene prioritising using an efficient and biologically appropriate network-based consensus strategy;Journal of Computational Science;2022-11
2. Activation of mitochondrial TRAP1 stimulates mitochondria-lysosome crosstalk and correction of lysosomal dysfunction;iScience;2022-09
3. Nitrosative stress in Parkinson’s disease;npj Parkinson's Disease;2022-08-11
4. Nitric oxide-based regulation of metabolism: Hints from TRAP1 and SIRT3 crosstalk;Frontiers in Molecular Biosciences;2022-07-26
5. TRAP1 in Oxidative Stress and Neurodegeneration;Antioxidants;2021-11-19
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