Mitochondria in the signaling pathways that control longevity and health span-Reference-Cited by-同舟云学术

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Mitochondria in the signaling pathways that control longevity and health span

Published:2019-09 Issue: Volume:54 Page:100940
ISSN:1568-1637
Container-title:Ageing Research Reviews
language:en
Short-container-title:Ageing Research Reviews

Author:

Akbari Mansour,Kirkwood Thomas B.L.,Bohr Vilhelm A.

Funder

NORDEA Foundation, Denmark

JPND

Innovation Fund Denmark

Olav Thon foundation Norway

Novo Nordisk foundation Denmark

Intramural Research Program of the NIH, National Institute on Aging, USA

Publisher

Elsevier BV

Subject

Neurology,Molecular Biology,Aging,Biochemistry,Biotechnology

Reference332 articles.

1. Cockayne syndrome group B protein promotes mitochondrial DNA stability by supporting the DNA repair association with the mitochondrial membrane;Aamann;FASEB J.,2010

2. Telomerase does not counteract telomere shortening but protects mitochondrial function under oxidative stress;Ahmed;J. Cell. Sci.,2008

3. Cytotoxicity and mutagenicity of endogenous DNA base lesions as potential cause of human aging;Akbari;Mech. Ageing Dev.,2008

4. Extracts of proliferating and non-proliferating human cells display different base excision pathways and repair fidelity;Akbari;DNA Repair (Amst.),2009

5. Overexpression of DNA ligase III in mitochondria protects cells against oxidative stress and improves mitochondrial DNA base excision repair;Akbari;DNA Repair (Amst.),2014

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