Advanced Glycation End Products Acutely Impair Ca2+ Signaling in Bovine Aortic Endothelial Cells-Reference-Cited by-同舟云学术

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Advanced Glycation End Products Acutely Impair Ca2+ Signaling in Bovine Aortic Endothelial Cells

Published:2013 Issue: Volume:4 Page:
ISSN:1664-042X
Container-title:Frontiers in Physiology
language:
Short-container-title:Front. Physiol.

Author:

Naser Nadim,Januszewski Andrzej S.,Brown Bronwyn E.,Jenkins Alicia J.,Hill Michael A.,Murphy Timothy V.

Publisher

Frontiers Media SA

Subject

Physiology (medical),Physiology

Cited by 19 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Microfluidic investigation for shear-stress-mediated repair of dysglycemia-induced endothelial cell damage;Mechanobiology in Medicine;2024-09

2. Advanced glycated end‐products inhibit dilation through constitutive endothelial RAGE and Nox1/4 in rat isolated skeletal muscle arteries;Microcirculation;2023-11-20

3. The Molecular Heterogeneity of Store-Operated Ca2+ Entry in Vascular Endothelial Cells: The Different roles of Orai1 and TRPC1/TRPC4 Channels in the Transition from Ca2+-Selective to Non-Selective Cation Currents;International Journal of Molecular Sciences;2023-02-07

4. Acute Exposure to Glycated Proteins Impaired in the Endothelium-Dependent Aortic Relaxation: A Matter of Oxidative Stress;International Journal of Molecular Sciences;2022-11-29

5. Modification of Fibronectin by Non-Enzymatic Glycation Impairs K+ Channel Function in Rat Cerebral Artery Smooth Muscle Cells;Frontiers in Physiology;2022-06-27

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