The Evolution of Cholesterol-Rich Membrane in Oxygen Adaption: The Respiratory System as a Model-Reference-Cited by-同舟云学术

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The Evolution of Cholesterol-Rich Membrane in Oxygen Adaption: The Respiratory System as a Model

Published:2019-10-29 Issue: Volume:10 Page:
ISSN:1664-042X
Container-title:Frontiers in Physiology
language:
Short-container-title:Front. Physiol.

Author:

Zuniga-Hertz Juan Pablo,Patel Hemal H.

Funder

Center for Integrated Healthcare, U.S. Department of Veterans Affairs

National Institutes of Health

Publisher

Frontiers Media SA

Subject

Physiology (medical),Physiology

Reference203 articles.

1. Functional and transcriptional induction of aquaporin-1 gene by hypoxia; analysis of promoter and role of Hif-1alpha.;Abreu-Rodriguez;PLoS One,2011

2. Arterial remodeling and plasma volume expansion in caveolin-1-deficient mice.;Albinsson;Am. J. Physiol. Regul. Integr. Comp. Physiol.,2007

3. Maximal oxygen consumption is reduced in aquaporin-1 knockout mice.;Al-Samir;Front. Physiol.,2016

4. Effect of cholesterol on the molecular structure and transitions in a clinical-grade lung surfactant extract.;Andersson;Proc. Natl. Acad. Sci. U.S.A.,2017

5. predicted decrease in membrane oxygen permeability with addition of cholesterol.;Angles;Adv. Exp. Med. Biol.,2017

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2. Glucose and Oxygen Levels Modulate the Pore-Forming Effects of Cholesterol-Dependent Cytolysin Pneumolysin from Streptococcus pneumoniae;Toxins;2024-05-21

3. A paREDOX in the control of cholesterol biosynthesis;BioEssays;2024-05-17

4. Sprouty1 is a broad mediator of cellular senescence;Cell Death & Disease;2024-04-26

5. Cholesterol inhibits oxygen permeation through biological membranes: mechanism against double-bond peroxidation;RSC Advances;2024

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