JNK Signaling Pathway Mediates Acetaminophen-Induced Hepatotoxicity Accompanied by Changes of Glutathione S-Transferase A1 Content and Expression-Reference-Cited by-同舟云学术

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JNK Signaling Pathway Mediates Acetaminophen-Induced Hepatotoxicity Accompanied by Changes of Glutathione S-Transferase A1 Content and Expression

Published:2019-09-20 Issue: Volume:10 Page:
ISSN:1663-9812
Container-title:Frontiers in Pharmacology
language:
Short-container-title:Front. Pharmacol.

Author:

Shi Chenxi,Hao Beili,Yang Yang,Muhammad Ishfaq,Zhang Yuanyuan,Chang Yicong,Li Ying,Li Changwen,Li Rui,Liu Fangping

Funder

National Natural Science Foundation of China

Publisher

Frontiers Media SA

Subject

Pharmacology (medical),Pharmacology

Reference49 articles.

1. Role of nitric oxide and reduced glutathione in the protective effects of aminoguanidine, gadolinium chloride and oleanolic acid against acetaminophen-induced hepatic and renal damage;Abdel-Zaher;Toxicology,2007

2. The effect of menadione on glutathione S-transferase A1 (GSTA1): c-Jun N-terminal kinase (JNK) complex dissociation in human colonic adenocarcinoma Caco-2 cells;Adnan;Toxicol. Lett.,2012

3. Chitohexaose protects against acetaminophen-induced hepatotoxicity in mice;Barman;Cell Death Dis.,2016

4. SP600125, an anthrapyrazolone inhibitor of Jun N-terminal kinase;Bennett;Proc. Natl. Acad. Sci. U. S. A.,2001

5. Acute liver failure;Bernal;N. Engl. J. Med.,2013

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3. Evaluation of the protective effect of losartan in acetaminophen-induced liver and kidney damage in mice;Naunyn-Schmiedeberg's Archives of Pharmacology;2024-01-09

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