How Mucosal Epithelia Deal with Stress: Role of NKG2D/NKG2D Ligands during Inflammation-Reference-Cited by-同舟云学术

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How Mucosal Epithelia Deal with Stress: Role of NKG2D/NKG2D Ligands during Inflammation

Published:2017-11-20 Issue: Volume:8 Page:
ISSN:1664-3224
Container-title:Frontiers in Immunology
language:
Short-container-title:Front. Immunol.

Author:

Antonangeli Fabrizio,Soriani Alessandra,Cerboni Cristina,Sciumè Giuseppe,Santoni Angela

Funder

Associazione Italiana per la Ricerca sul Cancro

Istituto Pasteur-Fondazione Cenci Bolognetti

Ministero dell’Istruzione, dell’Università e della Ricerca

Sapienza Università di Roma

Publisher

Frontiers Media SA

Subject

Immunology,Immunology and Allergy

Reference73 articles.

1. Costimulation through NKG2D enhances murine CD8+ CTL function: similarities and differences between NKG2D and CD28 costimulation;Markiewicz;J Immunol,2005

2. RAE1epsilon ligand expressed on pancreatic islets recruits NKG2D receptor-expressing cytotoxic T cells independent of T cell receptor recognition;Markiewicz;Immunity,2012

3. Coordinated induction by IL15 of a TCR-independent NKG2D signaling pathway converts CTL into lymphokine-activated killer cells in celiac disease;Meresse;Immunity,2004

4. Role of NKG2D signaling in the cytotoxicity of activated and expanded CD8+ T cells;Verneris;Blood,2004

5. Natural killer cell recognition of in vivo drug-induced senescent multiple myeloma cells;Antonangeli;Oncoimmunology,2016

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