Forward Genetics Reveals a gatC-gatA Fusion Polypeptide Causes Mistranslation and Rifampicin Tolerance in Mycobacterium smegmatis-Reference-Cited by-同舟云学术

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Forward Genetics Reveals a gatC-gatA Fusion Polypeptide Causes Mistranslation and Rifampicin Tolerance in Mycobacterium smegmatis

Published:2020-09-24 Issue: Volume:11 Page:
ISSN:1664-302X
Container-title:Frontiers in Microbiology
language:
Short-container-title:Front. Microbiol.

Author:

Cai Rong-Jun,Su Hong-Wei,Li Yang-Yang,Javid Babak

Funder

Bill and Melinda Gates Foundation

National Natural Science Foundation of China

Publisher

Frontiers Media SA

Subject

Microbiology (medical),Microbiology

Reference23 articles.

1. Molecular mechanism of aminoglycoside antibiotic kinase APH(3′)-IIIa: roles of conserved active site residues.;Boehr;J. Biol. Chem.,2001

2. Kasugamycin potentiates rifampicin and limits emergence of resistance in Mycobacterium tuberculosis by specifically decreasing mycobacterial mistranslation.;Chaudhuri;Elife,2018

3. Measurement of specific mycobacterial mistranslation rates with gain-of-function reporter systems.;Chen;J. Vis. Exp.,2019

4. Deciphering the biology of Mycobacterium tuberculosis from the complete genome sequence.;Cole;Nature,1998

5. Glu-tRNAGln amidotransferase: a novel heterotrimeric enzyme required for correct decoding of glutamine codons during translation.;Curnow;Proc. Natl. Acad. Sci. U.S.A.,1997

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