Dietary Zinc Supplementation Prevents Autism Related Behaviors and Striatal Synaptic Dysfunction in Shank3 Exon 13–16 Mutant Mice-Reference-Cited by-同舟云学术

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Dietary Zinc Supplementation Prevents Autism Related Behaviors and Striatal Synaptic Dysfunction in Shank3 Exon 13–16 Mutant Mice

Published:2018-10-22 Issue: Volume:12 Page:
ISSN:1662-5102
Container-title:Frontiers in Cellular Neuroscience
language:
Short-container-title:Front. Cell. Neurosci.

Author:

Fourie Chantelle,Vyas Yukti,Lee Kevin,Jung Yewon,Garner Craig C.,Montgomery Johanna M.

Funder

Marsden Fund

Health Research Council of New Zealand

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience

Reference71 articles.

1. Shank3 is part of a zinc-sensitive signaling system that regulates excitatory synaptic strength;Arons;J. Neurosci.,2016

2. Autism associated mutations in ProSAP2/Shank3 impair synaptic transmission and neurexin-neuroligin mediated transsynaptic signaling;Arons;J. Neurosci.,2012

3. Release of endogenous Zn2+ from brain tissue during activity;Assaf;Nature,1984

4. An architectural framework that may lie at the core of the postsynaptic density;Baron;Science,2006

5. ProSAP/Shank proteins-a family of higher order organizing molecules of the postsynaptic density with an emerging role in human neurological disease;Boeckers;J. Neurochem.,2002

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1. Unravelling the role of SHANK3 mutations in targeted therapies for autism spectrum disorders;Discover Psychology;2024-09-06

2. Differential effectiveness of dietary zinc supplementation with autism-related behaviours in Shank2 knockout mice;Philosophical Transactions of the Royal Society B: Biological Sciences;2024-06-10

3. Iron, magnesium, zinc and selenium - The most common elemental deficiencies in children with autism spectrum disorder;Research in Autism Spectrum Disorders;2024-02

4. Cellular zinc metabolism and zinc signaling: from biological functions to diseases and therapeutic targets;Signal Transduction and Targeted Therapy;2024-01-03

5. GluA1-Shank3 interaction decreases in response to chronic neuronal depolarization;Neuroscience Letters;2023-07

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