Affiliation:
1. Institute for Social Pediatrics and Adolescent Medicine, Ludwig-Maximilians-University of Munich, Haydnstr. 5, D-80336 München, Germany
Abstract
In recent decades, theories have been presented to explain the nature of dyslexia, but the causes of dyslexia remained unclear. Although the investigation of the causes of dyslexia presupposes a clear understanding of the concept of cause, such an understanding is missing. The present paper proposes the absence of at least one necessary condition or the absence of all sufficient conditions as causes for impaired reading. The causes of impaired reading include: an incorrect fixation location, too short a fixation time, the attempt to recognize too many letters simultaneously, too large saccade amplitudes, and too short verbal reaction times. It is assumed that a longer required fixation time in dyslexic readers results from a functional impairment of areas V1, V2, and V3 that require more time to complete temporal summation. These areas and areas that receive input from them, such as the fusiform gyrus, are assumed to be impaired in their ability to simultaneously process a string of letters. When these impairments are compensated by a new reading strategy, reading ability improves immediately.
Reference221 articles.
1. Berkhan, O. (1889). Über Störungen der Sprache und der Schriftsprache: Für Ärzte und Lehrer Dargestellt, August Hirschwald.
2. Über Dyslexie;Berlin;Arch. Psychiat. Nervenkrankh.,1884
3. Helland, T. (2022). Trends in dyslexia research during the period 1950 to 2020—Theories, definitions, and publications. Brain Sci., 12.
4. Stein, J. (2023). Theories about developmental dyslexia. Brain Sci., 13.
5. Physiological and anatomical evidence for a magnocellular defect in developmental dyslexia;Livingstone;Proc. Natl. Acad. Sci. USA,1991
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