Endogenous Selenoprotein P, a Liver-Derived Secretory Protein, Mediates Myocardial Ischemia/Reperfusion Injury in Mice
Author:
Publisher
MDPI AG
Subject
Inorganic Chemistry,Organic Chemistry,Physical and Theoretical Chemistry,Computer Science Applications,Spectroscopy,Molecular Biology,General Medicine,Catalysis
Link
http://www.mdpi.com/1422-0067/19/3/878/pdf
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2. Selenoprotein P deficiency protects against immobilization-induced muscle atrophy by suppressing atrophy-related E3 ubiquitin ligases;American Journal of Physiology-Endocrinology and Metabolism;2023-06-01
3. Emerging Evidence Linking the Liver to the Cardiovascular System: Liver-derived Secretory Factors;Journal of Clinical and Translational Hepatology;2023-05-12
4. Cardiac Hepatopathy: New Perspectives on Old Problems through a Prism of Endogenous Metabolic Regulations by Hepatokines;Antioxidants;2023-02-17
5. Safety of selenium exposure and limitations of selenoprotein maximization: Molecular and epidemiologic perspectives;Environmental Research;2022-08
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