3′,5′-Cyclic Adenosine Monophosphate Response Element-Binding Protein and CCAAT Enhancer-Binding Protein Are Dispensable for Insulin Inhibition of Phosphoenolpyruvate Carboxykinase Transcription and for Its Synergistic Induction by Protein Kinase A and Glucocorticoids
Author:
Affiliation:
1. Pennsylvania State University College of Medicine, Department of Cellular and Molecular Physiology, Hershey, Pennsylvania 17033
Publisher
The Endocrine Society
Subject
Endocrinology,Molecular Biology,General Medicine
Link
http://academic.oup.com/mend/article-pdf/19/4/913/8951197/mend0913.pdf
Reference66 articles.
1. Phosphoenolpyruvate carboxykinase. I. Its role in gluconeogenesis.;Hanson;Am J Clin Nutr,1972
2. Molecular physiology of the regulation of hepatic gluconeogenesis and glycolysis.;Pilkis;Annu Rev Physiol,1992
3. Phosphoenolpyruvate carboxykinase (GTP): the gene and the enzyme.;Hanson;Adv Enzymol Relat Areas Mol Biol,1994
4. Regulation of phosphoenolpyruvate carboxykinase (GTP) gene expression.;Hanson;Annu Rev Biochem,1997
5. cAMP stimulates transcription of the gene for cytosolic phosphoenolpyruvate carboxykinase in rat liver nuclei.;Lamers;Proc Natl Acad Sci USA,1982
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